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60,61,64 Additional studies indicate that BPC 157 supports VEGFR2 internalization and VEGFR2–Akt–eNOS activation, independent of shear stress or ligands, while also exerting context-specific control over angiogenesis, enhancing vascularization in ischemic muscle and tendon but suppressing pathological corneal neovascularization

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auris clinical strains (18,519, 1370, 3396, and 4574) it was obtained higher PICA% values at MIC/4 comparatively to MIC/2 demonstrating the efficacy of this concentration against adherence ability

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