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Necroptosis plays a dual role in tumour immune regulation, as it can activate antigen-specific immune responses by releasing damage-associated molecular patterns, while RIPK3/MLKL-mediated IL-1α release can suppress T-cell function and promote tumour immune escape [59]

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Certainly, the very extensive work of the group of Acuna-Castroviejo [179] is consistent with the high efficacy of melatonin in reducing the consequences of sepsis at the mitochondrial level