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On top of this, some cancer cells express receptors for GHRH itself- effectively a target for GHRH analogs like sermorelin

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Resistant phenotypes can arise as a result of increased pump activity due to their overexpression, as is observed with the addition of sublethal concentrations of antibiotics, which, through a cascade of feedback-driven interactions, causes an increase in the expression of MDR pump genes that evacuate these antibiotics [71]