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Laboratory studies evaluate kinase activation, protein synthesis regulation, cellular metabolism, intracellular communication, transcription factor activity, and coordinated molecular responses

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In metastatic breast cancer, tumor-infiltrating neutrophils (TINs) upregulate ACOD1 through the GM-CSF-JAK/STAT5-C/EBPβ pathway, producing itaconate, which activates an NRF2-dependent antiferroptotic program, sustaining TIN survival and immunosuppression