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Other reports indicate that excessive levels of NO, as a result, for instance, from BPC 157 activity, can be responsible for damage of mitochondrial iron–sulfur enzymes and hence for inhibition of mitochondrial respiration through inhibition of cytochrome oxidase [154,155]

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To clarify the relationship between pretreatment IGF-1 and response to GH, height velocity (SD) between groups with IGF-1 above (n = 34) and below (n = 26) the cutoff value (− 1.493 SD) was compared