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Nevertheless, pharmacological maintenance or restoration of GSH levels can inhibit NF-κB signaling and cytokine release, thereby preventing excessive or chronic inflammation.37 Thus, although the present IHC findings suggest possible modulation of NF-κB–related pathways, definitive mechanistic interpretation requires molecular-level confirmation through gene or protein expression analyses

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In this guideline, we do not use the term pernicious anaemia to describe autoimmune gastritis